Author

S. Boeing

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Casal· 1 shared A. Martinez-Riano· 1 shared B. Ludewig· 1 shared C. Bode· 1 shared D. Hoving· 1 shared I. V. Aramburu· 1 shared K. Klaus Stahl· 1 shared K. M. Spillane· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. Casal

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Long-term retention of antigens in germinal centres is controlled by the spatial organisation of the follicular dendritic cell network

A. Martinez-Riano, S. Wang, S. Boeing, S. Minoughan, A. Casal, K. M. Spillane, B. Ludewig, P. Tolar

The paper shows that follicular dendritic cells (FDCs) in germinal centers (GCs) have distinct spatial organization that controls antigen retention, with central FDCs acting as long-term antigen reservoirs.

bioRxiv · Sep 6, 2022 · PreprintImmunology

SIGNR1 promotes immune dysfunction in systemic candidiasis by modulating neutrophil lifespan via T cell-derived histones and G-CSF

M. Ioannou, D. Hoving, I. V. Aramburu, N. M. De Vasconcelos, M. I. Temkin, Q. Wang, S. Vernardis, V. Demichev, T.-D. Tsourouktsoglou, S. Boeing, R. Goldstone, S. David, K. Klaus Stahl, C. Bode, M. Ralser, V. Papagiannopoulos

This study identifies SIGNR1's role in promoting immune dysfunction during systemic candidiasis by modulating neutrophil lifespan through T cell-derived histones and G-CSF.

bioRxiv · Aug 9, 2021 · PreprintImmunology