Author

C. Plumlee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Aderem· 1 shared A. Diercks· 1 shared A. Jahn· 1 shared A. Lietzke· 1 shared A. Rothchild· 1 shared D. Mai· 1 shared E. Gold· 1 shared F. Duffy· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperImmunology

A. Aderem

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2 papers

Recent·Most cited

TOLLIP resolves lipid-induced EIF2 signaling in alveolar macrophages for durable Mycobacterium tuberculosis protection.

S. Venkatasubramanian, C. Plumlee, K. Dill-McFarland, G. Pearson, S. B. Cohen, A. Lietzke, S. A. Hinderstein, R. Pryor, S. A. Soleimanpour, M. Altman, K. B. Urdahl, J. A. Shah

This study shows TOLLIP prevents lipid-induced EIF2 activation in alveolar macrophages, which allows Mycobacterium tuberculosis replication and reduces protection.

bioRxiv · Aug 24, 2020 · PreprintImmunology

Latent Mycobacterium tuberculosis infection provides protection for the host by changing the activation state of the innate immune system

J. Nemeth, G. S. Olson, A. Rothchild, A. Jahn, D. Mai, F. Duffy, J. Delahaye, S. Srivatsan, C. Plumlee, K. Urdahl, E. Gold, A. Diercks, A. Aderem

This study shows that latent Mycobacterium tuberculosis infection protects hosts by altering the activation state of the innate immune system in both mice and humans.

bioRxiv · Feb 27, 2019Immunology