Author

G. Pearson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1

Frequent Co-Authors

A. A. Fitzgerald· 1 shared A. Lietzke· 1 shared A. Nasir· 1 shared A. R. Gorringe· 1 shared A. White· 1 shared C. Ho· 1 shared C. Kennard· 1 shared C. Plumlee· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperImmunology

A. A. Fitzgerald

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Fibroblast activation protein regulates natural killer cell migration, extravasation and tumor infiltration

A. A. Fitzgerald, R. E. Maynard, E. F. Marcisak, A. Nasir, E. Glasgow, S. Jablonski, P. Van der Veken, G. Pearson, S. E. Eisman, E. M. Mace, E. J. Fertig, L. M. Weiner

This study shows that fibroblast activation protein (FAP) is expressed by human NK cells and promotes their migration, extravasation, and tumor infiltration, suggesting FAP as a target to enhance NK cell-based therapies.

bioRxiv · Feb 3, 2021 · PreprintImmunology

Immunological and pathological outcomes of SARS-CoV-2 challenge after formalin-inactivated vaccine immunisation of ferrets and rhesus macaques

K. R. Bewley, K. E. R. Gooch, K. M. Thomas, S. R. Longet, N. Wiblin, L. Hunter, K. Chan, P. Brown, R. A. Russell, C. Ho, G. Slack, H. E. Humphries, L. Alden, L. Allen, M. Aram, , M. Carroll

This study evaluates vaccine-enhanced disease in ferrets and rhesus macaques after formalin-inactivated SARS-CoV-2 vaccination, finding mild transient lung pathology but no significant enhancement.

bioRxiv · Dec 21, 2020Pathology

TOLLIP resolves lipid-induced EIF2 signaling in alveolar macrophages for durable Mycobacterium tuberculosis protection.

S. Venkatasubramanian, C. Plumlee, K. Dill-McFarland, G. Pearson, S. B. Cohen, A. Lietzke, S. A. Hinderstein, R. Pryor, S. A. Soleimanpour, M. Altman, K. B. Urdahl, J. A. Shah

This study shows TOLLIP prevents lipid-induced EIF2 activation in alveolar macrophages, which allows Mycobacterium tuberculosis replication and reduces protection.

bioRxiv · Aug 24, 2020 · PreprintImmunology