Author

Carola G. Vinuesa

The Crick InstitutePrimary ORCID profile (0000-0001-9799-0298 · 3 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Carola G. VinuesaiD3 papers · The Francis Crick Institute

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

Adrienne Hilton· 1 shared Alexander G. BassukiD· 1 shared Allison CoxiD· 1 shared Anne Tripaydonis· 1 shared Ben T. Kile· 1 shared Benjamin W. DarbroiD· 1 shared Cameron D. Griffiths· 1 shared Carolyn A. de Graaf· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

Adrienne Hilton

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2 papers

Recent·Most cited

MicrobiologybioRxivPreprintNov 4, 2026

Lung Epithelial TLR7 is a Factor in Viral Replication and Pathogenesis

Madison J. Granoski, Ryley D. McClelland, Leanne M. Bilawchuk…·David J. Marchant

This study shows that TLR7 in lung epithelial cells drives RSV and influenza replication and disease, but not HPIV3, and links TLR7 expression to sex differences in viral disease.

DOI 10.64898/2026.11.03.764012

𝕏 PostBluesky

Cell BiologybioRxivPreprintMay 15, 2019

Missense mutations in the MLKL brace region lead to lethal neonatal inflammation in mice and are present in high frequency in humans.

Joanne M. Hildebrand, Maria Kauppi, Ian J. Majewski…·John Silke

This study identifies a missense mutation in MLKL causing lethal neonatal inflammation in mice and links it to increased frequency in human autoinflammatory disease.

DOI 10.1101/628370

𝕏 PostBluesky