Author

D. Feigenbutz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Mishra· 1 shared D. Hornburg· 1 shared F. Meissner· 1 shared I. Dudanova· 1 shared I. Riera-Tur· 1 shared L. Fernandez-Mosquera· 1 shared M. Mann· 1 shared N. Raimundo· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Mishra

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1 paper

Recent·Most cited

Amyloid-like aggregates cause lysosomal defects in neurons via gain-of-function toxicity

I. Riera-Tur, T. Schaefer, D. Hornburg, A. Mishra, L. Fernandez-Mosquera, D. Feigenbutz, P. Auer, M. Mann, W. Baumeister, R. Klein, F. Meissner, N. Raimundo, R. Fernandez-Busnadiego, I. Dudanova

This study shows that amyloid-like aggregates impair lysosomal function in neurons by sequestering AP-31, leading to neurotoxicity.

bioRxiv · Dec 16, 2019 · PreprintCell Biology