Author

D. J. Davidson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

D. Minns· 3 shared E. Gwyer Findlay· 2 shared K. J. Smith· 2 shared L. Melrose· 2 shared A. S. MacDonald· 1 shared A. Williams· 1 shared B. J. McHugh· 1 shared B. McHugh· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersImmunology

D. Minns

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

The antimicrobial peptide cathelicidin is critical for the development of Th17 responses in severe inflammatory disease

K. J. Smith, D. Minns, B. McHugh, R. Holloway, R. O'Connor, A. Williams, L. Melrose, R. McPherson, V. E. Miron, D. J. Davidson, E. Gwyer Findlay

This study shows cathelicidin promotes Th17 responses and severe EAE by enhancing T cell differentiation and plasticity, and its absence protects mice from disease.

bioRxiv · Jan 27, 2022 · PreprintImmunology

Classical macrophage polarisation is limited by human β-defensin 3 via an autocrine IL-4 dependent process.

M. E. Candela, D. J. P. Allsop, R. N. Carter, F. Semple, F. Kilanowski, S. Webb, D. Taggart, H. J. W. Mullan, B. J. McHugh, D. H. Dockrell, D. J. Davidson, J. E. Allen, S. J. Jenkins, N. M. Morton, J. R. Dorin

This paper shows HBD3 limits macrophage classical activation and promotes alternative activation via IL-4R, enabling a repair-conducive state.

bioRxiv · May 6, 2021 · PreprintImmunology

Ultra-pure isolation of low density neutrophils casts doubt on their exceptionality in health and disease

G. R. Hardisty, F. Llanwarne, D. Minns, J. L. Gillan, D. J. Davidson, E. R. Gwyer Findlay, R. D. Gray

The paper isolates ultra-pure low density neutrophils (LDNs) from healthy donors and shows they are functionally similar to normal density neutrophils (NDNs), challenging their classification as a distinct population.

bioRxiv · Jun 17, 2020Immunology

The neutrophil antimicrobial peptide cathelicidin promotes Th17 differentiation

D. Minns, K. J. Smith, V. Alessandrini, G. Hardisty, L. Melrose, L. H. Jackson-Jones, A. S. MacDonald, D. J. Davidson, E. Gwyer Findlay

This study shows cathelicidin promotes Th17 differentiation by enhancing AHR and RORγt expression, increasing IL-17F production, and protecting Th17 cells from apoptosis.

bioRxiv · Apr 10, 2020Immunology