Author

D. Sproul

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

A. Meynert· 1 shared D. Wilson· 1 shared E. Jarman· 1 shared G. Grimes· 1 shared J. C. Acosta· 1 shared K. Gournopanos· 1 shared L. Boulter· 1 shared M. Taylor· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Meynert

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1 paper

Recent·Most cited

In vivo modelling of patient genetic heterogeneity identifies concurrent Wnt and PI3K activity as a potent driver of invasive cholangiocarcinoma growth.

N. Younger, M. Wilson, E. Jarman, A. Meynert, G. Grimes, K. Gournopanos, S. Waddell, P. Tennant, D. Wilson, R. Guest, S. Wigmore, J. C. Acosta, T. J. Kendall, M. Taylor, D. Sproul, , L. Boulter

This study identifies concurrent Wnt and PI3K activity as drivers of invasive cholangiocarcinoma growth and shows that their co-inhibition slows tumor growth in vivo.

bioRxiv · Jul 5, 2021 · PreprintCancer Biology