Author

D. W. Nebert

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 1

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Thompson· 1 shared C. T. Shearn· 1 shared D. C. Thompson· 1 shared D. J. Orlicky· 1 shared E. A. Davidson· 1 shared G. Charkoftaki· 1 shared R. G. Kibbey· 1 shared R. L. Cardone· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

B. Thompson

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1 paper

Recent·Most cited

Endocrine pancreas-specific Gclc gene deletion causes a severe diabetes phenotype

E. A. Davidson, Y. Chen, S. Singh, D. J. Orlicky, B. Thompson, Y. Wang, G. Charkoftaki, T. Furnary, R. L. Cardone, R. G. Kibbey, C. T. Shearn, D. W. Nebert, D. C. Thompson, V. Vasiliou

This study shows that deleting the Gclc gene in the endocrine pancreas of mice leads to severe diabetes due to oxidative stress and islet dysfunction.

bioRxiv · Jun 13, 2023 · PreprintPhysiology