Author

E. Fraser

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Ecology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Antanaviciute· 1 shared A. Califano· 1 shared A. Kung· 1 shared A. M. Hale· 1 shared A. Obradovic· 1 shared C. Clelland· 1 shared C. G. Drake· 1 shared C. J. Campbell· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperImmunology

A. Antanaviciute

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Unusual migratory strategy a key factor driving interactions at wind energy facilities in at-risk bats

C. J. Campbell, D. M. Nelson, J. Nagel, J. Clerc, T. J. Weller, J. G. Weiringa, E. Fraser, F. J. Longstaffe, A. M. Hale, M. Lout, L. Pruitt, R. Guralnick, H. B. V. Zanden

The paper reveals a unique migratory strategy in bats that correlates with increased wind turbine fatalities, highlighting migratory distance as a key factor in interactions with wind energy facilities.

bioRxiv · Jan 28, 2024 · PreprintEcology

Case Study of Single-cell Protein Activity Based Drug Prediction for Precision Treatment of Cholangiocarcinoma

A. Obradovic, L. Tomassoni, D. Yu, K. Guillan, K. Souto, E. Fraser, S. Bates, C. G. Drake, Y. Saenger, F. D. Cruz, A. Kung, A. Califano

The paper develops a single-cell framework to prioritize drugs for cholangiocarcinoma by analyzing subpopulations and validating predictions in patient-derived xenografts.

bioRxiv · Feb 28, 2022 · PreprintCancer Biology

Type-1 IFN primed monocytes in pathogenesis of idiopathic pulmonary fibrosis

L.-P. Ho, E. Fraser, L. Denney, K. Blirando, C. Vuppusetty, A. Antanaviciute, Y. Zheng, E. Repapi, V. Iotchkova, S. Taylor, N. Ashley, V. St Noble, R. Hoyles, R. Benamore, C. Clelland, , J. Rehwinkel

The paper identifies type-1 IFN-primed monocytes as key drivers of fibrosis in IPF, showing their increased levels, altered phenotype, and potential as therapeutic targets.

bioRxiv · Jan 16, 2020 · PreprintImmunology