Author

E. Hsu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:zebrafish· 1

Frequent Co-Authors

A. J. Berk· 1 shared D. M. Bowles· 1 shared E. Scholl· 1 shared H. Soderholm· 1 shared H.-Y. Shih· 1 shared J. A. Wohlschlegel· 1 shared J. Bonkowsky· 1 shared J. Sha· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMicrobiology

A. J. Berk

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Author profile →

2 papers

Recent·Most cited

Adenovirus E1A Binding to DCAF10 Targets Degradation of AAA+ ATPases Required for Quaternary Assembly of Multiprotein Machines and Innate Immunity

N. R. Zemke, W. D. Barshop, J. Sha, E. Hsu, J. A. Wohlschlegel, A. J. Berk

The paper identifies that adenovirus E1A's C-terminal region interacts with host proteins to form a complex that degrades AAA+ ATPases, thereby inhibiting IRF3 stabilization and innate immune responses.

bioRxiv · Dec 16, 2020 · PreprintMicrobiology

Vanishing white matter disease expression of truncated EIF2B5 activates induced stress response

M. Keefe, H. Soderholm, H.-Y. Shih, T. Stevenson, K. Glaittli, D. M. Bowles, E. Scholl, S. Colby, S. Merchant, E. Hsu, J. Bonkowsky

This study shows that truncated EIF2B5 expression in zebrafish models of VWM activates the ISR and impairs motor behavior, suggesting a feed-forward mechanism in disease pathophysiology.

bioRxiv · May 5, 2020Physiology