Author

E. M. Hessel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Chandra· 1 shared A. G. Richter· 1 shared A. M. Condliffe· 1 shared A.-K. Stark· 1 shared E. Banham-Hall· 1 shared G. Bradley· 1 shared J. Clark· 1 shared J. E. Konkel· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperImmunology

A. Chandra

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintAug 4, 2022

Distinct eosinophil subsets are modulated by agonists of the commensal-metabolite and vitamin B3 receptor GPR109A during allergic-type inflammation

R. Azzoni, K. J. Filbey, R. H. Daw…·J. R. Grainger

The paper identifies GPR109A as a regulator of glycolytic Siglec-Fhi eosinophils during allergic inflammation, showing that GPR109A ligands reduce their frequency and activation.

DOI 10.1101/2022.08.04.502285

mousebulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivMar 18, 2018

PI3Kδ hyper-activation promotes the development of B cells that exacerbate Streptococcus pneumoniae infection in an antibody-independent manner

A.-K. Stark, A. Chandra, K. Chakraborty…·K. Okkenhaug

This study identifies a PI3Kδ-driven B cell subset in the lungs that worsens Streptococcus pneumoniae infection independently of antibodies, and shows that inhibiting PI3Kδ improves survival.

DOI 10.1038/s41467-018-05674-8

mouseexperimental assay

𝕏 PostBluesky