Author

E. Smit

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1 Research Focus:genome assembly· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Guislain· 1 shared A. Okonska· 1 shared A. van Hoeck· 1 shared A. Veenhof· 1 shared B. Nicolet· 1 shared C. Shale· 1 shared C. van Herpen· 1 shared E. Cuppen· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperImmunology

A. Guislain

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Tertiary lymphoid structure-related immune infiltrates in NSCLC tumor lesions correlate with low tumor-reactivity of TIL products

S. M. Castenmiller, N. Kanagasabesan, A. Guislain, B. Nicolet, M. van Loenen, K. Monkhorst, A. Veenhof, E. Smit, K. Hartemink, J. Haanen, R. de Groot, M. Wolkers

This study identifies that high B cell infiltrates in NSCLC tumors, associated with tertiary lymphoid structures, negatively correlate with the tumor-reactivity of expanded TIL products.

bioRxiv · Feb 19, 2024Immunology

Pan-cancer whole genome analyses of metastatic solid tumors

P. Priestley, J. Baber, M. Lolkema, N. Steeghs, E. de Bruijn, K. Duyvesteyn, S. Haidari, A. van Hoeck, W. Onstenk, P. Roepman, C. Shale, M. Voda, H. Bloemendal, V. Tjan-Heijnen, C. van Herpen, , E. Cuppen

This study analyzes whole genome sequences of metastatic solid tumors to identify genetic variants that may influence treatment outcomes.

bioRxiv · Aug 12, 2019 · PreprintCancer Biology

Genome-wide silencing screen in mesothelioma cells reveals that loss of function of BAP1 induces chemoresistance to ribonucleotide reductase inhibition: implication for therapy

A. Okonska, S. Buehler, V. Rao, M. Ronner, M. Blijlevens, I. Van der Meulen-Muileman, R. de Menezes, E. Smit, W. Weder, R. Stahel, L. Penengo, V. van Beusechem, E. Felley-Bosco

The paper identifies BAP1's role in regulating RRM2 during replication stress, showing that BAP1 loss increases chemoresistance to ribonucleotide reductase inhibitors in mesothelioma cells.

bioRxiv · Jul 31, 2018Cancer Biology