Author

F. Walper

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Research Focus:cell type annotation· 1 Organism:mouse· 1

Frequent Co-Authors

C. Drosten· 2 shared C. Goffinet· 2 shared D. Niemeyer· 2 shared J. Kazmierski· 2 shared J. Trimpert· 2 shared L. M. Jeworowski· 2 shared M. L. Schmidt· 2 shared S. Schroeder· 2 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersImmunology

C. Drosten

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Antigenic cartography using variant-specific hamster sera reveals substantial antigenic variation among Omicron subvariants

B. Mühlemann, J. Trimpert, F. Walper, M. L. Schmidt, S. Schroeder, L. M. Jeworowski, J. Beheim-Schwarzbach, T. Bleicker, D. Niemeyer, J. M. Adler, R. M. Vidal, C. Langner, D. Vladimirova, D. J. Smith, M. Voss, , C. Drosten

The paper uses hamster sera from variant-specific infections to map antigenic differences among SARS-CoV-2 variants, revealing significant antigenic variation among Omicron subvariants.

bioRxiv · Jul 2, 2023Immunology

HIV-1 infection causes depletion of monocytic cells through a non-canonical cell death pathway

M. Stuck, N. D. Arnow, J. Kazmierski, F. Walper, C. Goffinet, N. Bannert, O. Cingoz

This study shows HIV-1 infection causes monocytic cell death via a non-canonical pathway independent of nucleic acid sensing or IFN signaling, requiring viral gene products and occurring after integration.

bioRxiv · Dec 20, 2022 · PreprintMicrobiology

Post-entry, spike-dependent replication advantage of B.1.1.7 and B.1.617.2 over B.1 SARS-CoV-2 in an ACE2-deficient human lung cell line

D. Niemeyer, S. Schroeder, K. Friedmann, F. Weege, J. Trimpert, A. Richter, S. Stenzel, J. Jansen, J. Emanuel, J. Kazmierski, F. Pott, L. M. Jeworowski, R. Olmer, M.-C. Jaboreck, B. Tenner, , C. Drosten

The paper shows that B.1.1.7 and B.1.617.2 SARS-CoV-2 variants have a replication advantage over B.1 in ACE2-deficient lung cells, suggesting reliance on alternative cellular factors.

bioRxiv · Oct 20, 2021 · PreprintMicrobiology