Author

Frauke Stölting

Düsseldorf University Hospital

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

Christian Ballmeyer· 2 shared Gabriel LeprivieriD· 2 shared Georg FluegeniD· 2 shared Guido ReifenbergeriD· 2 shared Jonathan K. M. LimiD· 2 shared Marc RemkeiD· 2 shared Abhimanyu DasGupta· 1 shared Alberto DelaidelliiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

Christian Ballmeyer

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 23, 2026

GALE-dependent glycoproteome remodelling is a determinant of oncogenic RAS transformation

Frauke Stölting, Rita Vesce, César Rodríguez-Santana…·Jonathan K. M. Lim

This study identifies GALE as a key regulator of glycoproteome remodelling in RAS-driven cancer, showing its role in sialylation and tumorigenicity.

DOI 10.64898/2026.06.15.732364

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJan 27, 2024

Oncogenic RAS signaling is a tumor cell-intrinsic determinant of ferroptosis suppression via induction of the GCH1/BH4 axis

Jonathan K. M. Lim, Frauke Stölting, Dennis Roth…·Gabriel Leprivier

This study shows oncogenic RAS signaling suppresses ferroptosis by upregulating GCH1 and BH4, making RAS-driven cancers resistant to ferroptosis-inducing agents.

1 citation

humanbulk RNA-seq

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