Author

Jonathan K. M. Lim

Düsseldorf University HospitalORCID 0000-0002-1798-8954Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1

Frequent Co-Authors

Gabriel LeprivieriD· 3 shared Alberto DelaidelliiD· 2 shared Christian Ballmeyer· 2 shared Frauke Stölting· 2 shared Georg FluegeniD· 2 shared Guido ReifenbergeriD· 2 shared Marc RemkeiD· 2 shared Poul H. B. SorenseniD· 2 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersCancer Biology

Gabriel LeprivierORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 23, 2026

GALE-dependent glycoproteome remodelling is a determinant of oncogenic RAS transformation

Frauke Stölting, Rita Vesce, César Rodríguez-Santana…·Jonathan K. M. Lim

This study identifies GALE as a key regulator of glycoproteome remodelling in RAS-driven cancer, showing its role in sialylation and tumorigenicity.

DOI 10.64898/2026.06.15.732364

humanproteomics

𝕏 PostBluesky

Molecular BiologyCell Death and DiseasePeer-reviewedJul 13, 2024

The eEF2 kinase coordinates the DNA damage response to cisplatin by supporting p53 activation

Jonathan K. M. Lim, Arash Samiei, Alberto Delaidelli…·Poul H. B. Sorensen

This study shows eEF2K is critical for the DNA damage response to cisplatin, promoting p53 activation and DNA repair depending on damage severity.

13 citations

humanC. elegans

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Cancer BiologybioRxivPreprintJan 27, 2024

Oncogenic RAS signaling is a tumor cell-intrinsic determinant of ferroptosis suppression via induction of the GCH1/BH4 axis

Jonathan K. M. Lim, Frauke Stölting, Dennis Roth…·Gabriel Leprivier

This study shows oncogenic RAS signaling suppresses ferroptosis by upregulating GCH1 and BH4, making RAS-driven cancers resistant to ferroptosis-inducing agents.

1 citation

humanbulk RNA-seq

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