Author

G. Cannino

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Rasola· 2 shared F. Ciscato· 2 shared I. Masgras· 2 shared A. Castegna· 1 shared A. Filippi· 1 shared A. M. Brunati· 1 shared A. Menga· 1 shared A. Negro· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

A. Rasola

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologybioRxivDec 6, 2021

TRAP1 and cyclophilin D compete at OSCP subunit to regulate enzymatic activity and permeability transition pore opening by F-ATP synthase

G. Cannino, A. Urbani, M. Gaspari…·A. Rasola

DOI 10.1038/s41418-022-01020-0

experimental assaycell signaling & trafficking

𝕏 PostBluesky

Cancer BiologybioRxivMay 29, 2021

Tumor growth of neurofibromin-deficient cells is driven by decreased respiration and hampered by NAD+ and SIRT3

I. Masgras, G. Cannino, F. Ciscato…·A. Rasola

This study shows that neurofibromin loss reduces mitochondrial respiration and NAD+ levels, promoting tumor growth, and that restoring NAD+ or SIRT3 can counteract this effect.

DOI 10.1038/s41418-022-00991-4

humanmetabolomics

𝕏 PostBluesky