Author

G. D. Richardson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Walaszczyk· 2 shared E. Dookun· 2 shared H. Arthur· 2 shared J. Chapman· 2 shared J. F. Passos· 2 shared J. Mialet-Perez· 2 shared S. Tual-Chalot· 2 shared W. A. Owens· 2 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

A. Walaszczyk

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintApr 28, 2020

Clearance of senescent cells during cardiac ischemia-reperfusion injury improves heart recovery following myocardial infarction

E. Dookun, A. Walaszczyk, R. Redgrave…·G. D. Richardson

This study shows that clearing senescent cells after cardiac ischemia-reperfusion injury improves heart function, reduces scar size, and decreases inflammation.

DOI 10.1101/2020.04.28.065789

mouseproteomics

𝕏 PostBluesky

Cell BiologybioRxivAug 17, 2018

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani…·J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

DOI 10.15252/embj.2018100492

humanmouse

𝕏 PostBluesky