Author

G. Fajardo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Biophysics· 1

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Dawood· 1 shared A. Jhingran· 1 shared A. S. Vander Roest· 1 shared B. Pruitt· 1 shared C. Liu· 1 shared D. Bernstein· 1 shared D. Song· 1 shared G. Jung· 1 shared

Activity by Year

2020-2020 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperBiophysics

A. Dawood

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1 paper

Recent·Most cited

Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super-relaxed state: multiscale measurements and computational modeling

A. S. Vander Roest, C. Liu, M. M. Morck, K. B. Kooiker, G. Jung, D. Song, A. Dawood, A. Jhingran, G. Pardon, S. Ranjbarvaziri, G. Fajardo, M. Zhao, K. S. Campbell, B. Pruitt, J. A. Spudich, , D. Bernstein

This study investigates the P710R mutation in β-cardiac myosin, showing it causes hypercontractility by disrupting the super-relaxed state, using multiscale experiments and modeling.

bioRxiv · Nov 10, 2020 · PreprintBiophysics