Author

S. Ranjbarvaziri

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:imaging· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Budan· 1 shared A. Dawood· 1 shared A. Jhingran· 1 shared A. S. Vander Roest· 1 shared B. Pruitt· 1 shared C. Liu· 1 shared D. Bernstein· 1 shared D. Song· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Budan

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2 papers

Recent·Most cited

Cell BiologybioRxivMar 23, 2021

Deep Learning Predicts Patterns of Cardiotoxicity in a High-Content Screen Using Induced Pluripotent Stem Cell-Derived Cardiomyocytes

F. Grafton, J. Ho, S. Ranjbarvaziri…·M. A. Mandegar

This paper uses deep learning and high-content screening of iPSC-derived cardiomyocytes to predict cardiotoxicity patterns in 1280 compounds and identify cardiotoxic chemical frameworks.

DOI 10.7554/elife.68714

humanimaging

𝕏 PostBluesky

BiophysicsbioRxivPreprintNov 10, 2020

Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super-relaxed state: multiscale measurements and computational modeling

A. S. Vander Roest, C. Liu, M. M. Morck…·D. Bernstein

This study investigates the P710R mutation in β-cardiac myosin, showing it causes hypercontractility by disrupting the super-relaxed state, using multiscale experiments and modeling.

DOI 10.1101/2020.11.10.375493

humanproteomics

𝕏 PostBluesky