Author

A. Jhingran

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biophysics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Assay:proteomics· 2 Approach:statistical model· 2 Research Focus:clustering· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Klopp· 2 shared G. Biegert· 2 shared L. Colbert· 2 shared M. Ahmed-Kaddar· 2 shared T. Karpinets· 2 shared T. Solley· 2 shared T. T. Sims· 2 shared X. Wu· 2 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

A. Klopp

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super-relaxed state: multiscale measurements and computational modeling

A. S. Vander Roest, C. Liu, M. M. Morck, K. B. Kooiker, G. Jung, D. Song, A. Dawood, A. Jhingran, G. Pardon, S. Ranjbarvaziri, G. Fajardo, M. Zhao, K. S. Campbell, B. Pruitt, J. A. Spudich, , D. Bernstein

This study investigates the P710R mutation in β-cardiac myosin, showing it causes hypercontractility by disrupting the super-relaxed state, using multiscale experiments and modeling.

bioRxiv · Nov 10, 2020 · PreprintBiophysics

During Aspergillus infection, neutrophil, monocyte-derived DC, and plasmacytoid DC enhance innate immune defense through CXCR3-dependent crosstalk

Y. Guo, S. Kasahara, A. Jhingran, N. L. Tosini, B. Zhai, M. A. Aufiero, K. A. M. Mills, M. Gjonbalaj, V. Espinosa, A. Rivera, A. D. Luster, T. Hohl

The paper shows that pDCs enhance innate immune defense against Aspergillus by amplifying neutrophil activity through CXCR3-dependent crosstalk during infection.

bioRxiv · May 5, 2020Immunology

Adaptive changes in the gut microbiome during standard-of-care chemoradiotherapy for gynecologic cancers

M. B. El Alam, T. T. Sims, R. Kouzy, G. Biegert, J. Abi Jaoude, T. Karpinets, K. Y. Court, X. Wu, A. D. Medrano, M. Mezzari, N. Ajami, T. Solley, M. Ahmed-Kaddar, L. Lin, L. Ramondetta, , L. Colbert

This study tracks gut microbiome diversity and composition changes in gynecologic cancer patients undergoing chemoradiotherapy, finding reduced diversity during treatment but partial recovery by week 12.

bioRxiv · Apr 10, 2020Cancer Biology

Dynamic evolution of cervical cancer mutations during chemoradiation using novel sampling approach

B. V. Chapman, T. Karpinets, T. T. Sims, G. Biegert, X. Wu, A. Y. Delgado Medrano, P. J. Eifel, A. Jhingran, L. L. Lin, L. M. Ramondetta, A. M. Futreal, A. A. Jazaeri, M. Frumovitz, K. M. Schmeler, J. Yue, , L. Colbert

This study uses whole exome sequencing to track cervical cancer mutations during chemoradiation, finding that CDK4/CDK6/cyclin D1-related mutations persist or increase, suggesting radiation resistance.

bioRxiv · Nov 25, 2019 · PreprintCancer Biology