Author

G. Pompilio

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

A. S. Maione· 2 shared A. Zaza· 1 shared C. Florindi· 1 shared C. Maniezzi· 1 shared D. Pereira-Sousa· 1 shared D. Potesil· 1 shared D. Rovina· 1 shared E. Sacco· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

A. S. Maione

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

An iPSC-derived bio-inspired scaffold modelling the structure and the effects of extracellular matrix in cardiac fibrosis

F. Niro, S. Fernandes, M. Cassani, M. Apostolico, J. Oliver-De La Cruz, D. Pereira-Sousa, S. Pagliari, V. Vinarsky, Z. Zdrahal, D. Potesil, V. Pustka, G. Pompilio, E. Sommariva, D. Rovina, A. S. Maione, , G. Forte

This paper develops a bio-inspired scaffold from iPSC-derived cardiac myofibroblasts to model fibrotic ECM effects on cardiomyocytes.

bioRxiv · Feb 7, 2024 · PreprintCell Biology

Early consequences of the phospholamban mutation PLN-R14del+/- in a transgenic mouse model

C. Maniezzi, M. Eskandr, C. Florindi, M. Ferrandi, P. Barassi, E. Sacco, V. Pasquale, A. S. Maione, G. Pompilio, V. Oliveira Nunes Teixeira, R. A. de Boer, H. H. W. Sillje, F. Lodola, A. Zaza

This study examines Ca2+ dynamics and energy metabolism in a transgenic mouse model of the PLN-R14del mutation before cardiomyopathy onset, finding altered mitochondrial function and ER-stress activation.

bioRxiv · Apr 5, 2023Physiology