Author

H. B. Sager

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Dutsch· 1 shared A. Friebe· 1 shared A. Kopczak· 1 shared A. Liesz· 1 shared C. Fürle· 1 shared C. Mauersberger· 1 shared D. Brough· 1 shared DEMDAS Study Group· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. Dutsch

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Author profile →

2 papers

Recent·Most cited

ImmunologybioRxivPreprintFeb 1, 2023

Stroke induces early recurrent vascular events by inflammasome-dependent atherosclerotic plaque rupture

J. Cao, S. Roth, S. Zhang…·A. Liesz

This study shows stroke triggers inflammasome-dependent atherosclerotic plaque rupture, leading to recurrent stroke, and identifies DNA-mediated inflammasome activation as a therapeutic target.

DOI 10.1101/2023.02.01.526550

mouseexperimental assay

PhysiologybioRxivNov 15, 2021

Loss of soluble guanylyl cyclase in platelets contributes to atherosclerotic plaque formation and vascular inflammation

C. Mauersberger, H. B. Sager, J. Wobst…·T. Kessler

This study shows that loss of soluble guanylyl cyclase in platelets promotes atherosclerosis by reducing angiopoietin-1 release and increasing leukocyte adhesion, suggesting sGC stimulation as a potential therapy for coronary artery disease.

DOI 10.1038/s44161-022-00175-w

mouseexperimental assay