Author

I. Scionti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

L. Schaeffer· 2 shared A. Hamiche· 1 shared A. Osseni· 1 shared A. Ravel-Chapuis· 1 shared B. J. Jasmin· 1 shared C. Papin· 1 shared D. Dalkara· 1 shared E. Belotti· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersPhysiology

L. Schaeffer

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Author profile →

2 papers

Recent·Most cited

PhysiologybioRxivPreprintJan 21, 2022

Pharmacological inhibition of HDAC6 downregulates TGF-β via Smad2/3 acetylation and improves dystrophin-deficient muscles.

A. Osseni, A. Ravel-Chapuis, I. Scionti…·L. Schaeffer

This study shows that inhibiting HDAC6 with tubastatin A improves dystrophin-deficient muscle by reducing fibrosis and restoring microtubules and NMJ through Smad2/3 acetylation and TGF-β signaling inhibition.

DOI 10.1101/2022.01.21.477182

mouseexperimental assay

𝕏 PostBluesky

Cell BiologybioRxivOct 30, 2019

H2A.Z is dispensable for both basal and activated transcription in post-mitotic mouse muscles

E. Belotti, N. Lacoste, T. Simonet…·L. Schaeffer

This study shows H2A.Z is not required for transcription in post-mitotic muscle cells, challenging its role as an activator and suggesting it is a marker of active promoters.

DOI 10.1093/nar/gkaa157

mouseATAC-seq

𝕏 PostBluesky