Author

J. Farup

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

A. B. Moeller· 1 shared C. Comert· 1 shared E. Groppa· 1 shared E. Masschelein· 1 shared F. de Paoli· 1 shared F. M. V. Rossi· 1 shared G. Fitzgerald· 1 shared G. Turiel· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. B. Moeller

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

MME+ fibro-adipogenic progenitors are the dominant adipogenic population during fatty infiltration in human skeletal muscle

G. Fitzgerald, G. Turiel, T. Gorski, I. Soro-Arnaiz, J. Zhang, N. C. Casartelli, E. Masschelein, N. A. Maffiuletti, R. Sutter, M. Leunig, J. Farup, K. De Bock

The paper identifies MME+ fibro-adipogenic progenitors as the main adipogenic population in fatty infiltrated human skeletal muscle using single-cell RNA sequencing.

bioRxiv · Apr 22, 2022Cell Biology

Human skeletal muscle CD90+ fibro-adipogenic progenitors are associated with muscle degeneration in type 2 diabetes patients

J. Farup, J. Just, F. de Paoli, L. Lin, J. B. Jensen, T. Billeskov, I. S. Roman, C. Comert, A. B. Moeller, L. Madaro, E. Groppa, R. G. Fred, U. Kampmann, S. B. Pedersen, P. Bross, , N. Jessen

This study identifies CD90+ fibro-adipogenic progenitors in T2DM skeletal muscle and shows PDGF signaling drives their pathogenic behavior, which is mitigated by Metformin.

bioRxiv · Aug 25, 2020Cell Biology