Author

J. Oblong

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

J. D. Sherrill· 2 shared L. Vires· 2 shared M. Rodrigues· 2 shared R. L. Adams· 2 shared R. Li· 2 shared A. Acharjee· 1 shared A. Sanz· 1 shared B. Carroll· 1 shared

Activity by Year

2020-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersBiochemistry

J. D. Sherrill

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Author profile →

3 papers

Recent·Most cited

The vitamin A ester retinyl propionate has a unique metabolic profile and higher retinoid-related bioactivity over retinol and retinyl palmitate in human skin models.

D. Bjerke, R. Li, J. M. Price, R. Dobson, M. Rodrigues, C. Tey, L. Vires, R. L. Adams, J. D. Sherrill, P. B. Styczynski, K. Goncalves, V. Maltman, S. Przyborski, J. Oblong

This study compares the metabolic profiles and bioactivity of retinol, retinyl palmitate, and retinyl propionate in human skin models, finding RP has higher bioactivity and unique metabolism.

bioRxiv · Sep 26, 2020Biochemistry

Enhanced retinoid response by a combination of the vitamin A ester retinyl propionate with niacinamide and a flavonoid containing Ceratonia siliqua extract in retinoid responsive in vitro models.

J. Oblong, E. Lam, R. Li, M. Rodrigues, L. Vires, R. L. Adams, J. D. Sherrill

The paper evaluates if combining retinyl propionate with niacinamide and a Ceratonia siliqua extract enhances retinoid activity in vitro, finding increased gene expression and reporter activation.

bioRxiv · Sep 26, 2020Biochemistry

Autophagy promotes cell and organismal survival by maintaining NAD(H) pools

L. Sedlackova, E. G. Otten, F. Scialo, D. Shapira, T. Kataura, B. Carroll, E. Seranova, Y. Rabanal Ruiz, G. Kelly, R. Stefanatos, G. Nelson, F. Urselli, A. Acharjee, N. S. Kenneth, S. A. Trushin, , V. I. Korolchuk

This study identifies autophagy's role in maintaining NAD(H) pools, showing that NAD(H) depletion leads to cell death and that NAD(H) supplementation can reverse this effect in autophagy-deficient models.

bioRxiv · Jan 31, 2020 · PreprintCell Biology