Author

K. A. Paris

University of Pittsburgh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Research Focus:protein folding & dynamics· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

C. J. Camacho· 2 shared U. Santiago· 2 shared Carlos Jaime CamachoiD· 1 shared D. S. Patel· 1 shared David A. LewisiD· 1 shared Gregg E. HomanicsiD· 1 shared M L MacDonald· 1 shared Megan E. Garver· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersBiophysics

C. J. Camacho

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3 papers

Recent·Most cited

NeuroscienceMolecular PsychiatryPeer-reviewedFeb 1, 2021

MAP2 is differentially phosphorylated in schizophrenia, altering its function

Melanie J. Grubisha, X. Sun, M L MacDonald…·Robert A. Sweet

This study identifies MAP2 phosphorylation changes in schizophrenia, linking them to neuronal structure and synaptic function impairments through phosphoproteomics and transgenic mouse models.

38 citations

humanproteomics

𝕏 PostBluesky

BiophysicsbioRxivPreprintJul 29, 2020

On the molecular mechanism of SARS-CoV-2 retention in the upper respiratory tract

K. A. Paris, U. Santiago, C. J. Camacho

This study identifies a pH-dependent receptor binding mechanism in SARS-CoV-2 that enhances ACE2 receptor retention, leading to prolonged viral shedding and transmission.

DOI 10.1101/2020.07.29.227389

humanproteomics

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BiophysicsbioRxivPreprintJun 16, 2020

Loss of pH switch unique to SARS-CoV2 supports unfamiliar virus pathology

C. J. Camacho, K. A. Paris, U. Santiago

This paper analyzes the pH-switch difference between SARS-CoV1 and SARS-CoV2, showing how the loss of this switch in CoV2 allows prolonged ACE2 receptor binding, contributing to higher transmission and pathogenicity.

DOI 10.1101/2020.06.16.155457

humanstatistical model

𝕏 PostBluesky