Author

K. Adea

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Organism:rat· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

M. Bekliz· 3 shared M. Essaidi-Laziosi· 3 shared C. Alvarez· 2 shared C. Drosten· 2 shared I. Eckerle· 2 shared L. Kaiser· 2 shared P. Sattonnet-Roche· 2 shared A. Breithaupt· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMicrobiology

M. Bekliz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Immune escape and replicative capacity of Omicron lineages BA.1, BA.2, BA.5.1, BQ.1, XBB.1.5, EG.5.1 and JN.1.1

M. Bekliz, M. Essaidi-Laziosi, K. Adea, K. Hosszu-Fellous, C. Alvarez, M. Bellon, P. Sattonnet-Roche, O. Puhach, D. Dbeissi, M. E. Zaballa, S. Stringhini, I. Guessous, P. Vetter, C. S. Eberhardt, L. Kaiser, I. Eckerle

This study evaluates immune escape and replicative capacity of Omicron variants in human airway epithelial cells and sera from diverse immunological backgrounds.

bioRxiv · Feb 14, 2024 · PreprintMicrobiology

Distinct phenotype of SARS-CoV-2 Omicron BA.1 in human primary cells but no increased host range in cell lines of putative mammalian reservoir species

M. Essaidi-Laziosi, F. J. Perez Rodriguez, C. Alvarez, P. Sattonnet-Roche, G. Torriani, M. Bekliz, K. Adea, M. Lenk, W. Preiser, T. Suliman, M. A. Muller, C. Drosten, L. Kaiser, I. Eckerle

The paper compares the replicative capacity of SARS-CoV-2 variants in human primary cells and mammalian cell lines, finding Omicron BA.1 has a distinct phenotype in human nasal epithelia but no increased host range in other species.

bioRxiv · Oct 4, 2022Microbiology

Enhanced fitness of SARS-CoV-2 variant of concern B.1.1.7, but not B.1.351, in animal models

L. Ulrich, N. J. Halwe, A. Taddeo, N. Ebert, J. Schön, C. Devisme, B. S. Trüeb, B. Hoffmann, M. Wider, M. Bekliz, M. Essaidi-Laziosi, M. L. Schmidt, D. Niemeyer, V. M. Corman, A. Kraft, , C. Benarafa

The paper compares the fitness of SARS-CoV-2 variants B.1.1.7 and B.1.351 in animal models, finding B.1.1.7 has a fitness advantage in ferrets and mice but not in hamsters.

bioRxiv · Jun 28, 2021Microbiology