Author

K. Girdhar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Assay:proteomics· 3 Assay:metagenomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Raisingani· 2 shared E. ALTINDIS· 2 shared Q. Huang· 2 shared A. Bhansali· 1 shared A. Choubey· 1 shared A. K. Kar· 1 shared B. Medhi· 1 shared C. Brady· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersImmunology

A. Raisingani

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A Gut Microbial Peptide and Molecular Mimicry in the Pathogenesis of Type 1 Diabetes

K. Girdhar, Q. Huang, I.-T. Chow, C. Brady, A. Raisingani, P. Autissier, M. A. Atkinson, W. W. Kwok, C. R. Kahn, E. ALTINDIS

This paper identifies a gut microbial peptide that mimics an insulin epitope and contributes to Type 1 Diabetes pathogenesis by triggering immune cross-reactivity and accelerating disease onset in mice.

bioRxiv · Oct 22, 2020Immunology

Dynamics of Gut Microbiome, IgA Response and Plasma Metabolome in Development of Pediatric Celiac Disease

K. Girdhar, Q. Huang, Y. D. Dogru, Y. Yang, V. Tolstikov, M. Chrudinova, A. Raisingani, J. F. Ludvigsson, M. A. Kiebish, N. W. Palm, J. Ludvigsson, E. ALTINDIS

This study identifies distinct gut microbiota, IgA responses, and plasma metabolite changes in children who develop celiac disease before diagnosis.

bioRxiv · Feb 29, 2020 · PreprintImmunology

Hyperinsulinemia promotes HMGB1 release leading to inflammation induced systemic insulin resistance: An interplay between pancreatic beta-cell and peripheral organs

A. Choubey, A. K. Kar, K. Girdhar, T. Chattopadhyay, S. Dogra, S. Kushwaha, B. Medhi, A. Bhansali, C. K. Mantri, U. K. Seetharam, D. Ghosh, P. Mondal

This study shows hyperinsulinemia promotes inflammation via HMGB1 and TLR4 signaling, leading to systemic insulin resistance, and demonstrates naltrexone's protective effects.

bioRxiv · Jul 17, 2019 · PreprintPhysiology