Author

K. Yamagata

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Organism:mouse· 2 Assay:ChIP-seq· 1

Frequent Co-Authors

Y. Sato· 2 shared H. Kimura· 1 shared H. Kurumizaka· 1 shared H. Nishihara· 1 shared K. Suke· 1 shared M. Nakao· 1 shared M. Yamazaki· 1 shared N. Goto· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

Y. Sato

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell BiologybioRxivOct 20, 2023

ISWI chromatin remodeling complexes recruit NSD2 and H3K36me2 in pericentromeric heterochromatin

N. Goto, K. Suke, N. Yonezawa…·H. Kimura

The paper identifies NSD2 and ISWI chromatin remodeling complexes as mediators of H3K36me2 enrichment in pericentromeric heterochromatin and shows its presence in early mouse embryos.

DOI 10.1083/jcb.202310084

mouseChIP-seq

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Cancer BiologyThe EMBO JournalPeer-reviewedApr 25, 2023

ROR1 plays a critical role in pancreatic tumor-initiating cells with a partial EMT signature.

M. Yamazaki, S. Hino, S. Usuki…·K. Yamagata

This study identifies ROR1 as a critical marker for pancreatic tumor-initiating cells with a partial EMT signature, showing its role in promoting tumor growth and relapse.

DOI 10.15252/embj.2022112614

humanbulk RNA-seq

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BiochemistrybioRxivPreprintJun 28, 2022

Hypoxia causes pancreatic β-cell dysfunction by activating a transcriptional repressor BHLHE40

T. Tsuyama, Y. Sato, T. Yoshizawa…·K. Yamagata

This study identifies BHLHE40 as a hypoxia-induced transcriptional repressor that suppresses insulin secretion in pancreatic β-cells, contributing to β-cell dysfunction in type 2 diabetes.

DOI 10.1101/2022.06.28.498031

mousehuman

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