Author

K.-Y. A. Kim

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. A. Sarshad· 1 shared A. E. Murmann· 1 shared A. Haluck-Kangas· 1 shared A. M. Orriols· 1 shared A. Vassilopoulos· 1 shared C. Yau· 1 shared C. Yeh· 1 shared D. Horiuchi· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. A. Sarshad

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 21, 2023

S100A8/A9 predicts triple-negative breast cancer response to PIM kinase and PD-1/PD-L1 inhibition

L. R. Begg, A. M. Orriols, M. Zannikou…·D. Horiuchi

DOI 10.1101/2023.09.21.558870

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivSep 14, 2017

Many si/shRNAs can kill cancer cells by targeting multiple survival genes through an off-target mechanism

W. Putzbach, Q. Q. Gao, M. Patel…·M. E. Peter

The paper shows that many si/shRNAs targeting CD95 or CD95L induce cancer cell death via an off-target mechanism called DISE by eliminating survival genes.

DOI 10.7554/elife.29702

humanCRISPR screen

𝕏 PostBluesky