Author

L. Blaas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. V. Ritchie· 1 shared E. Kaasinen· 1 shared F. Zhong· 1 shared H. Cojoc· 1 shared I. Sur· 1 shared J. Dohla· 1 shared J. I. Englund· 1 shared J. Ivaska· 1 shared

Activity by Year

2016-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. V. Ritchie

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Author profile →

2 papers

Recent·Most cited

Laminin alpha 5 is Necessary for Mammary Epithelial Growth and Function by Maintaining Luminal Epithelial Cell Identity

J. I. Englund, H. Cojoc, L. Blaas, A. V. Ritchie, N. Pentinmikko, J. Dohla, P. Munne, M. Patarroyo, J. Klefstrom, J. Ivaska, P. Katajisto

This study shows that laminin alpha 5 is essential for mammary gland growth and function by maintaining luminal epithelial cell identity during development and remodeling.

bioRxiv · Dec 27, 2019 · PreprintCell Biology

Mice deficient of Myc super-enhancer region reveal a differential control mechanism between normal and pathological growth

K. Dave, I. Sur, J. Yan, J. Zhang, E. Kaasinen, F. Zhong, L. Blaas, X. Li, R. Mansson, J. Taipale

This study shows that deleting the Myc super-enhancer region in mice reduces Myc expression and tumor susceptibility without overt phenotypes, suggesting therapeutic potential for targeting this element in cancer.

bioRxiv · Oct 24, 2016Cancer Biology