Author

L. Ramos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Hamiche· 1 shared B. Zhai· 1 shared C. C. Collins· 1 shared C. Papin· 1 shared D. Brown· 1 shared D. Dalkara· 1 shared E. Belotti· 1 shared F. Ghaidi· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

A. Hamiche

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A bi-functional PARP-HDAC inhibitor with activity in Ewing sarcoma

L. Ramos, S. Truong, B. Zhai, J. Joshi, F. Ghaidi, M. M. Lizardo, T. Shyp, H. Adomat, S. LeBihan, C. C. Collins, J. Bacha, D. Brown, J. Langlands, W. Shen, N. Lallous, , M. Daugaard

This paper develops a dual PARP and HDAC inhibitor, kt-3283, showing enhanced cytotoxicity and efficacy in Ewing sarcoma models compared to single-agent inhibitors.

bioRxiv · Nov 10, 2022 · PreprintCancer Biology

H2A.Z is dispensable for both basal and activated transcription in post-mitotic mouse muscles

E. Belotti, N. Lacoste, T. Simonet, C. Papin, K. Padmanabhan, L. Ramos, D. Dalkara, I. Scionti, A. Hamiche, S. Dimitrov, L. Schaeffer

This study shows H2A.Z is not required for transcription in post-mitotic muscle cells, challenging its role as an activator and suggesting it is a marker of active promoters.

bioRxiv · Oct 30, 2019Cell Biology