Author

M. Collard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Assay:bulk RNA-seq· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2

Frequent Co-Authors

R. M. Alani· 3 shared K. Leibowitz· 2 shared N. Grbic· 2 shared P. A. Cole· 2 shared W. A. Wyant· 2 shared A. Hanly· 1 shared A. Labadorf· 1 shared A. R. Waddell· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersCancer Biology

R. M. Alani

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

p300 KAT regulates SOX10 stability and function in human melanoma

A. Waddell, N. Grbic, K. Leibowitz, W. A. Wyant, S. Choudhury, K. Park, M. Collard, P. A. Cole, R. M. Alani

This study identifies p300 KAT as a regulator of SOX10 stability and function in melanoma, showing that inhibiting p300 reduces SOX10 levels and melanoma proliferation and invasion.

bioRxiv · Feb 20, 2024Cancer Biology

The p300 KAT inhibitor, A-485, inhibits melanogenesis and melanin content in human melanocytes

N. Grbic, A. R. Waddell, K. Leibowitz, W. A. Wyant, M. Collard, R. M. Alani

This study shows that the p300 inhibitor A-485 reduces melanin production in human melanocytes by decreasing MITF and DCT expression.

bioRxiv · Feb 14, 2024 · PreprintCell Biology

HIV-induced membraneless organelles orchestrate post-nuclear entry steps.

V. Scoca, R. Morin, M. Collard, J.-Y. Tinevez, F. Di Nunzio

This study identifies HIV-induced membraneless organelles that facilitate viral replication by reorganizing the nuclear environment and promoting reverse transcription.

bioRxiv · Nov 17, 2020Microbiology

The CoREST Repressor Complex Mediates Phenotype Switching and Therapy Resistance in Melanoma

M. Wu, A. Hanly, F. Gibson, K. H. Kuang, J. Kalin, S. Nocco, M. Collard, M. W. Cole, A. Xiao, F. Agus, A. Labadorf, P. A. Cole, R. M. Alani

This study identifies the CoREST repressor complex as a mediator of melanoma phenotype switching and therapy resistance, showing that its inhibitor corin can reverse resistance to BRAF inhibitors.

bioRxiv · Sep 30, 2020Cancer Biology