M. van der Laan

This study shows that immature dengue virus particles activate the TLR2 axis in PBMCs, driving inflammation and endothelial activation, with TLR2 playing a role in both initiating and resolving inflammation.

This study reveals the structural role of the Mic60-Mic19 complex in forming mitochondrial crista junctions by acting as a molecular strut that controls their architecture.

This study identifies sialokinin in Aedes mosquito saliva as a factor that enhances arbovirus infection by reducing endothelial barrier integrity, suggesting it as a therapeutic target.