Author

Mark Turmaine

University College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1 Organism:zebrafish· 1

Frequent Co-Authors

Hugo CabedoiD· 2 shared Jose Antonio Gomez-SancheziD· 2 shared Peter Arthur‐FarrajiD· 2 shared Shaline V. FazaliD· 2 shared Agueda A. RostagnoiD· 1 shared Alastair Morris KilpatrickiD· 1 shared André StrydomiD· 1 shared Andrea LoretoiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersNeuroscience

Hugo CabedoORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintDec 8, 2022

SARM1 detection in oligodendrocytes but not Schwann cells though sarm1/Sarm1 deletion does not perturb CNS nor PNS myelination in zebrafish and mice.

Shaline V. Fazal, Clara Mutschler, Civia Zi-Yu Chen…·Peter Arthur‐Farraj

The paper shows SARM1 is present in oligodendrocytes but not Schwann cells, and its deletion does not affect myelination in zebrafish or mice.

DOI 10.1101/2022.12.08.519209

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NeuroscienceeLifePeer-reviewedJan 21, 2021

Failures of nerve regeneration caused by aging or chronic denervation are rescued by restoring Schwann cell c-Jun

Laura J. Wagstaff, Jose Antonio Gomez-Sanchez, Shaline V. Fazal…·Kristján R. Jessen

The paper shows that restoring Schwann cell c-Jun rescues nerve regeneration failures in aging and chronic denervation by enhancing regenerative support.

133 citationstop 1%

mouseexperimental assay

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NeuroscienceMolecular PsychiatryPeer-reviewedJul 9, 2020

Patient-specific Alzheimer-like pathology in trisomy 21 cerebral organoids reveals BACE2 as a gene-dose-sensitive AD-suppressor in human brain

Ivan Alić, Pollyanna Agnes Goh, Aoife Murray…·Henrik Zetterberg

The paper shows that trisomy 21 cerebral organoids exhibit AD-like pathology, which can be suppressed by BACE2 trisomy and inhibited by BACE1 inhibitors, suggesting BACE2 as an AD-suppressor gene.

123 citationstop 1%

humanproteomics

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