Author

N. Khodayari

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Assay:proteomics· 3

Frequent Co-Authors

M. Brantly· 3 shared R. Oshins· 3 shared B. Mehrad· 2 shared J. Lascano· 2 shared x. qiang· 2 shared C. Eagan· 1 shared G. Aslanidi· 1 shared G. Wiesemann· 1 shared

Activity by Year

2018-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersCell Biology

M. Brantly

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3 papers

Recent·Most cited

Cell BiologybioRxivJul 7, 2022

Correlation of Alpha-1 Antitrypsin Levels and Exosome Associated Neutrophil Elastase Endothelial Injury in Subjects with SARS-CoV2 Infection

J. Lascano, R. Oshins, C. Eagan…·N. Khodayari

The paper investigates the correlation between exosome-associated neutrophil elastase (NE) and alpha-1 antitrypsin levels in SARS-CoV-2 patients, finding that exosome-associated NE is resistant to alpha-1 antitrypsin inhibition and correlates with endothelial injury.

DOI 10.1371/journal.pone.0274427

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivJul 7, 2022

Cigarette smoke exposed airway epithelial cell-derived EVs promote pro-inflammatory macrophage activation in alpha-1 antitrypsin deficiency

N. Khodayari, R. Oshins, B. Mehrad…·M. Brantly

This study shows that cigarette smoke-exposed airway epithelial cell-derived EVs enhance pro-inflammatory activation of AATD macrophages, increasing cytokine production and neutrophil chemoattractant release.

DOI 10.1186/s12931-022-02161-z

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivDec 2, 2018

Neutrophil Elastase Activates Macrophage MMPs, Promotes Cell Adhesion And Cytokine Production Via Integrin-Src Kinases Pathway

K. Krotova, N. Khodayari, R. Oshins…·M. Brantly

This study shows that neutrophil elastase activates macrophages via integrin-Src kinases, increasing MMP activity, cytokine production, and cell adhesion, contributing to tissue destruction in diseases like cystic fibrosis.

DOI 10.1038/s41598-020-72667-3

humanproteomics

𝕏 PostBluesky