Author

O. Bedard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

E. Forte· 2 shared N. Rosenthal· 2 shared A. Chiramel· 1 shared A. Shamsaddini· 1 shared C. Baker· 1 shared C. Brunton· 1 shared C. Clancy· 1 shared C. Martens· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersMicrobiology

E. Forte

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2 papers

Recent·Most cited

Genetically diverse mouse models of SARS-CoV-2 infection reproduce clinical variation in type I interferon and cytokine responses in COVID-19

S. Robertson, O. Bedard, K. McNally, C. Shaia, C. Clancy, M. Lewis, R. Broeckel, A. Chiramel, J. G. Shannon, G. Sturdevant, R. Rosenke, S. L. Anzick, E. Forte, C. Preuss, C. Baker, , S. Best

The paper uses genetically diverse mice to model varied innate immune responses to SARS-CoV-2 infection, showing differences in survival, viral replication, and cytokine profiles.

bioRxiv · Sep 17, 2021Microbiology

Myocardial damage induced by a single high dose of isoproterenol in C57BL/6J mice triggers a persistent adaptive immune response against the heart.

E. Forte, M. Panahi, F. S. Ng, J. J. Boyle, J. Branca, O. Bedard, M. G. Hasham, L. Benson, S. E. Harding, N. Rosenthal, S. Sattler

This study shows that a single high dose of isoproterenol in mice causes myocardial damage and a persistent adaptive immune response against the heart, making it a suitable model for studying anti-heart autoimmunity.

bioRxiv · Feb 27, 2020Immunology