Author

R. C. Dash

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Vindigni· 1 shared D. J. Rowlands· 1 shared E. Berthiaume· 1 shared J. Calabrese· 1 shared J. Calvo· 1 shared J. Jackson· 1 shared K. Cong· 1 shared K. Hadden· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Vindigni

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2 papers

Recent·Most cited

Development of Enterovirus anti-viral agents that target the viral 2C protein

R. Kejriwal, T. Evans, J. Calabrese, L. Swistak, L. Alexandrescu, M. Cohen, N. Rahman, N. Henriksen, R. C. Dash, K. Hadden, N. J. Stonehouse, D. J. Rowlands, N. J. Kingston, M. Hartnoll, S. Dobson, S. White

This study develops broad-spectrum anti-viral compounds targeting the conserved EV 2C protein, showing low IC50 values and suggesting a role for 2C in capsid assembly.

bioRxiv · Oct 6, 2022Pharmacology & Toxicology

Replication gaps are a cancer vulnerability counteracted by translesion synthesis

S. Nayak, J. Calvo, K. Cong, E. Berthiaume, J. Jackson, R. C. Dash, A. Vindigni, K. M. Hadden, S. Cantor

The paper identifies that oncogene expression creates replication gaps, which are suppressed by translesion synthesis (TLS), and shows that inhibiting TLS reduces cancer cell fitness.

bioRxiv · Sep 25, 2019Cancer Biology