Author

R. Espin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Assay:CRISPR screen· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

M. A. Pujana· 2 shared A. Bernat· 1 shared A. Figueras· 1 shared A. Lahiguera· 1 shared A. Malik· 1 shared A. Villanueva· 1 shared D. Schramek· 1 shared D. Trcka· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

M. A. Pujana

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2 papers

Recent·Most cited

A rise in double-strand breaks sensitizes tumours to oxidative metabolism inhibitors

F. Medina-Jover, A. Figueras, A. Lahiguera, P. Guillen, R. Espin, M. A. Pardo, M. A. Pujana, E. Berra, A. Villanueva, A. Bernat, M. Romeo, J. C. Perales, F. Vinals

The paper shows that double-strand breaks (DSBs) induce a metabolic shift towards oxidative metabolism in tumors, making them more sensitive to oxidative metabolism inhibitors.

bioRxiv · Dec 19, 2023 · PreprintCancer Biology

Loss of epigenetic regulation disrupts lineage integrity, reactivatesmultipotency and promotes breast cancer

E. Langille, K. N. Al-Zahrani, Z. Ma, A. Malik, S. K. Loganathan, D. Trcka, J. Liu, K. Kozma, R. Tsai, K. Teng, R. Espin, S. Barutcu, T. Nguyen, R. Bremner, H. W. Jackson, , D. Schramek

This study identifies epigenetic regulators as tumor suppressors in breast cancer by showing their loss leads to lineage infidelity and reactivation of lactation programs.

bioRxiv · Oct 22, 2021 · PreprintCancer Biology