Author

R. K. Ma

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

P. Sethupathy· 3 shared K. Vakili· 2 shared M. Kanke· 2 shared A. B. Francisco· 1 shared A. Emili· 1 shared A. J. Shumway· 1 shared A. Karki· 1 shared A. P. Massa· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersCancer Biology

P. Sethupathy

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3 papers

Recent·Most cited

Cancer BiologybioRxivJun 2, 2023

DNAJB1-PRKACA fusion protein-regulated LINC00473 promotes tumor growth and alters mitochondrial fitness in fibrolamellar carcinoma

R. K. Ma, P.-Y. Tsai, A. R. Farghli…·P. Sethupathy

This paper identifies LINC00473 as a downstream mediator of DNAJB1-PRKACA fusion in fibrolamellar carcinoma, showing it promotes tumor growth and alters mitochondrial function.

DOI 10.1371/journal.pgen.1011216

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivAug 11, 2021

DNAJB1-PRKACA in HEK293T cells induces LINC00473 overexpression that depends on PKA signaling

S. S. Kim, I. Kycia, M. Karski…·K. Vakili

The paper engineers a DNAJB1-PRKACA fusion in HEK293T cells to study its oncogenic effects, finding LINC00473 overexpression dependent on PKA signaling.

DOI 10.1371/journal.pone.0263829

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJan 18, 2020

Hotspots of aberrant enhancer activity in fibrolamellar carcinoma reveal molecular mechanisms of oncogenesis and intrinsic drug resistance

T. A. Dinh, R. Sritharan, F. D. Smith…·P. Sethupathy

This study identifies aberrant enhancer activity in fibrolamellar carcinoma, revealing molecular mechanisms of oncogenesis and drug resistance, and suggests new therapeutic targets.

DOI 10.1101/2020.01.18.911297

humanChIP-seq

𝕏 PostBluesky