Author

S. M. Rubin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 3 Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:gene regulatory network inference· 2 Assay:proteomics· 2 Approach:statistical model· 2 Approach:biophysical modeling· 1

Frequent Co-Authors

A. N. Iness· 1 shared B. R. Topacio· 1 shared C. S. Tambo· 1 shared E. T. Wick· 1 shared E. Zatulovskiy· 1 shared J. E. Purvis· 1 shared J. Felthousen· 1 shared J. M. Skotheim· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. N. Iness

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell BiologybioRxivApr 23, 2021

Cyclin F drives proliferation through SCF-dependent degradation of the retinoblastoma-like tumor suppressor p130/RBL2

T. P. Enrico, W. Stallaert, E. T. Wick…·M. J. Emanuele

This study identifies cyclin F as a regulator of p130/RBL2 degradation via SCF, showing its role in cell cycle control and potential implications in cancer.

DOI 10.7554/elife.70691

humanproteomics

𝕏 PostBluesky

Cancer BiologyOncogenePeer-reviewedSep 11, 2018

The cell cycle regulatory DREAM complex is disrupted by high expression of oncogenic B-Myb

A. N. Iness, J. Felthousen, V. Ananthapadmanabhan…·L. Litovchick

This study shows that B-Myb disrupts the DREAM complex by affecting LIN52 protein levels and DYRK1A kinase activity, contributing to cancer progression.

DOI 10.1038/s41388-018-0490-y

humanbulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivAug 21, 2018

Cyclin D-Cdk4,6 drives cell cycle progression via the retinoblastoma protein’s C-terminal helix

B. R. Topacio, E. Zatulovskiy, S. Cristea…·J. M. Skotheim

This study shows cyclin D-Cdk4,6 drives cell cycle progression by phosphorylating Rb's C-terminal helix, which is distinct from other cyclin-Cdk interactions and critical for tumor suppression.

DOI 10.1016/j.molcel.2019.03.020

humanproteomics

𝕏 PostBluesky