Author

Simon F. Roy

Primary ORCID profile (0000-0002-5444-0349 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Simon F. RoyiD1 paper · Institut du cancer Rosalind & Morris Goodman

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

Afsar Barlas· 1 shared Elisa de StanchinaiD· 1 shared Eric Chan· 1 shared Hongyan Li· 1 shared Jacob A. BoyeriD· 1 shared Katia Manova-Todorova· 1 shared Malvika SharmaiD· 1 shared Marcus Bosenberg· 1 shared

Activity by Year

2023-2023 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Afsar Barlas

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1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintNov 21, 2023

Tumorigenesis driven by the BRAF V600E oncoprotein requires secondary mutations that overcome its feedback inhibition of migration and invasion

Sunyana Gadal, Jacob A. Boyer, Simon F. Roy…·Neal Rosen

This study shows that BRAFV600E mutations inhibit cell migration and invasion through feedback inhibition of RAC1, requiring secondary mutations to overcome this block and enable tumorigenesis.

DOI 10.1101/2023.11.21.568071

humanDNA methylation

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