Author

T. Sherrill

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

J. A. Kropski· 3 shared T. S. Blackwell· 3 shared A. S. McCall· 2 shared C. L. Calvi· 2 shared D. Nichols· 2 shared J. J. Gokey· 2 shared A. Burman· 1 shared A. J. Gutierrez· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersCell Biology

J. A. Kropski

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Author profile →

3 papers

Recent·Most cited

Hypoxia-inducible factor-2 (HIF2) regulates alveolar regeneration after repetitive injury

A. S. McCall, H. Tanjore, A. Burman, T. Sherrill, M. Chapman, C. L. Calvi, J. Camarata, R. P. Hunt, D. Nichols, N. Banovich, W. E. Lawson, J. J. Gokey, J. A. Kropski, T. S. Blackwell

Inhibiting hypoxia-inducible-factor 2 (HIF2) promotes functional lung alveolar epithelial repair following recurrent injury.

bioRxiv · Sep 17, 2023 · PreprintCell Biology

Epithelial Yap/Taz are required for functional alveolar regeneration following acute lung injury

G. T. DiGiovanni, W. Han, T. Sherrill, C. J. Taylor, D. S. Nichols, N. M. Geis, U. K. Singha, C. L. Calvi, A. S. McCall, M. M. Dixon, Y. Lui, J.-H. Jang, S. S. Gutor, V. V. Polosukhin, T. S. Blackwell, , J. J. Gokey

This study shows that Yap/Taz in AT2 cells are essential for alveolar regeneration and preventing fibrosis after lung injury.

bioRxiv · Jun 22, 2023Cell Biology

Secretory cells are the primary source of pIgR in small airways

J. B. Blackburn, J. A. Schaff, S. Gutor, R.-H. Du, D. Nichols, T. Sherrill, A. J. Gutierrez, M. K. Xin, N. Wickersham, Y. Zhang, M. J. Holtzman, L. B. Ware, N. E. Banovich, J. A. Kropski, T. S. Blackwell, B. W. Richmond

The paper identifies secretory cells as the main source of pIgR in airways and shows reduced pIgR protein expression, not cell numbers, contributes to SIgA loss in COPD.

bioRxiv · Nov 10, 2021Cell Biology