Author

Travis C. Lantz

Stanford Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Molecular Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Craig H. KerriD· 2 shared Davide RuggeroiD· 2 shared Maria BarnaiD· 2 shared Alakesh AlakeshiD· 1 shared Alma L. BurlingameiD· 1 shared Andrew G. LevineiD· 1 shared Carolyn K. JonsiD· 1 shared Craig M. ForesteriD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMolecular Biology

Craig H. KerrORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pharmacology & ToxicologyACS Pharmacology & Translational SciencePeer-reviewedSep 23, 2025

Intranasal sertraline for the investigation of nose-to-brain delivery to mitigate systemic exposure

Shoshana C. Williams, Travis C. Lantz, Vanessa M. Doulames…·Eric Andrew Appel

This study develops intranasal sertraline formulations to reduce systemic exposure while maintaining brain delivery, showing comparable brain exposure at half the dose compared to oral administration in mice.

2 citations

mouseexperimental assay

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Molecular BiologyMolecular CellPeer-reviewedSep 1, 2024

RAPIDASH: A tag-free enrichment of ribosome-associated proteins reveals compositional dynamics in embryonic tissues and stimulated macrophages

Teodorus Theo Susanto, Victoria Hung, Andrew G. Levine…·Maria Barna

RAPIDASH enriches ribosome-associated proteins from diverse samples, revealing their roles in neurodevelopment and immune response.

23 citations

mouseproteomics

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Developmental BiologyDevelopmental CellPeer-reviewedJul 1, 2021

A p53-dependent translational program directs tissue-selective phenotypes in a model of ribosomopathies

Gerald C. Tiu, Craig H. Kerr, Craig M. Forester…·Maria Barna

This study identifies a p53-dependent translational program that explains tissue-selective phenotypes in ribosomopathies by regulating 4E-BP1 and altering the translatome.

59 citations

mousestatistical model

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