Author

V. Dolinsky

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

G. Hatch· 2 shared A. Ravandi· 1 shared A. Srivastava· 1 shared B. Xiang· 1 shared C. Clark· 1 shared C. Doucette· 1 shared C. Pasco· 1 shared D. Schibli· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBioengineering

G. Hatch

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A multi-omics analysis of glioma chemoresistance using a hybrid microphysiological model of glioblastoma

S. Shojaei, J. Basso, M. Amereh, J. Alizadeh, T. Dehesh, S. D. S. Rosa, C. Clark, M. Hassan, M. Tomczyk, L. Cole, G. Hatch, V. Dolinsky, C. Pasco, D. Schibli, S. Dhingra, , M. Akbari

This study uses a hybrid microphysiological model to analyze glioma chemoresistance and identifies molecular changes associated with resistance mechanisms.

bioRxiv · Oct 29, 2022 · PreprintBioengineering

The Cardiolipin Transacylase Tafazzin Regulates Basal Insulin Secretion and Mitochondrial Function in Pancreatic Islets from Mice

L. K. Cole, P. Agarwal, C. Doucette, M. Fonseca, B. Xiang, G. C. Sparagna, N. Seshadri, M. Vandel, V. Dolinsky, G. Hatch

This study shows that Tafazzin deficiency in pancreatic islets reduces insulin secretion and mitochondrial function under low-glucose conditions.

bioRxiv · Jan 15, 2021Physiology