Author

Y. Honda

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 2 Organism:human· 2 Approach:Bayesian inference· 1 Organism:C. elegans· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Nawata· 1 shared A. Sugimoto· 1 shared B. Lin· 1 shared C. Uchiyama· 1 shared E. Hiejima· 1 shared E. Sumiyoshi· 1 shared H. Kataoka· 1 shared H. Nihira· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 9 mutual co-author links

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1 shared paperPathology

A. Nawata

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivMay 14, 2023

Bayesian Approach Enabled Objective Comparison of Multiple Human iPSC-derived Cardiomyocytes' Proarrhythmia Sensitivities.

T. Wakatsuki, N. Daily, S. Hisada…·K. Takasuna

The paper uses Bayesian statistics to objectively compare proarrhythmic sensitivities of five human iPSC-derived cardiomyocyte lines to ten compounds, identifying differences in calcium transient responses.

DOI 10.1016/j.vascn.2024.107531

humanelectrophysiology

𝕏 PostBluesky

Cell BiologybioRxivPreprintDec 23, 2022

Germline-specific role for unconventional components of the γ-tubulin complex in Caenorhabditis elegans

N. Haruta, E. Sumiyoshi, Y. Honda…·A. Sugimoto

This study identifies GTAP-1 and GTAP-2 as unconventional γ-tubulin complex components in C. elegans that target the complex to centrosomes and germ-cell membranes, affecting microtubule organization.

DOI 10.1101/2022.12.23.521729

C. elegansgenetic perturbation / CRISPR

𝕏 PostBluesky

ImmunologybioRxivSep 3, 2021

Aberrant localization of CDC42 C-terminal variants to the Golgi apparatus drives pyrin inflammasome-dependent autoinflammation

M. Nishitani-Isa, K. Mukai, Y. Honda…·T. Yasumi

This study identifies that CDC42 C-terminal variants cause aberrant Golgi localization, leading to pyrin inflammasome activation and autoinflammation.

DOI 10.1084/jem.20211889

humanproteomics

𝕏 PostBluesky

PathologybioRxivDec 4, 2020

Effect of belt electrode-skeletal muscle electrical stimulation on immobilization-induced muscle fibrosis

Y. Honda, N. Tanaka, Y. Kajiwara…·M. Okita

This study investigates how belt electrode-skeletal muscle electrical stimulation (B-SES) affects immobilization-induced muscle fibrosis by reducing macrophage accumulation and fibrotic markers in rats.

DOI 10.1371/journal.pone.0244120

ratexperimental assay

𝕏 PostBluesky