Author

Bilyana Stoilova

University of OxfordORCID 0000-0001-6192-9145Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

Batchimeg UsukhbayariD· 2 shared Marlen MetzneriD· 2 shared Mirian Angulo Salazar· 2 shared Niels Asger JakobseniD· 2 shared Paresh VyasiD· 2 shared Sven TurkaljiD· 2 shared Adrian H. Taylor· 1 shared Alexander Murison· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Batchimeg UsukhbayarORCID

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2 papers

Recent·Most cited

Cancer BiologyBloodPeer-reviewedNov 5, 2025

Rapid clonal selection within early hematopoietic cell compartments presages outcome to ivosidenib combination therapy

Sven Turkalj, Bilyana Stoilova, Angus Groom…·Paresh Vyas

This study shows that resistant clones are rapidly selected in hematopoietic cell compartments with leukemic stem cell potential before relapse in patients treated with ivosidenib combination therapy.

5 citations

humanscRNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintSep 12, 2023

Selective advantage of mutant stem cells in clonal hematopoiesis occurs by attenuating the deleterious effects of inflammation and aging

Niels Asger Jakobsen, Sven Turkalj, Andy G. X. Zeng…·Paresh Vyas

This study identifies how DNMT3A and TET2 mutations in hematopoietic stem cells confer a selective advantage by reducing inflammation and aging effects.

DOI 10.1101/2023.09.12.557322

humanscRNA-seq

𝕏 PostBluesky