Author

Christopher S. Nabel

Harvard UniversityORCID 0000-0001-6515-8579Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:DNA methylation· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

Matthew G. Vander HeideniD· 2 shared A. A. I. DeikiD· 1 shared Aaron N. HataiD· 1 shared Ahmed AliiD· 1 shared Ajinkya S. KawaleiD· 1 shared Alexander MuiriD· 1 shared Bradley I. ReinfeldiD· 1 shared Caroline A. LewisiD· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersMolecular Biology

Matthew G. Vander HeidenORCID

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2 papers

Recent·Most cited

Molecular BiologyMolecular CellPeer-reviewedApr 29, 2024

Unprocessed Genomic Uracil as a Source of DNA Replication Stress in Cancer Cells

Sneha Saxena, Christopher S. Nabel, Turner W. Seay…·Lee Zou

The paper shows genomic uracil induces DNA replication stress without BER processing and identifies UNG2 upregulation as a vulnerability in cancer cells.

41 citationstop 2%

DNA methylationexperimental assay

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Cancer BiologyeLifePeer-reviewedFeb 13, 2024

Metabolite profiling of human renal cell carcinoma reveals tissue-origin dominance in nutrient availability

Keene L. Abbott, Ahmed Ali, Bradley I. Reinfeld…·Matthew G. Vander Heiden

This study profiles metabolites in renal cell carcinoma tissue and fluid to show that nutrient availability is more influenced by tissue-specific factors than tumor-driven changes.

24 citations

humanmetabolomics

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