Author

Kate Hawkins

University College LondonORCID 0000-0001-6666-4238Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Anna Louise DavidiD· 1 shared Catherine Caruso· 1 shared Ellen PetzendorferiD· 1 shared Filipa Vlahova· 1 shared Fleur Stephanie van DijkiD· 1 shared Janet BrownleesiD· 1 shared Jill C. RichardsoniD· 1 shared Katie Durrant· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

Anna Louise DavidORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintMar 25, 2025

Rejuvenated human amniotic fluid stem cells: a superior source of standardized induced mesenchymal stem cells for enhanced therapeutic applications

Michelangelo Corcelli, Ellen Petzendorfer, Kate Hawkins…·Pascale V. Guillot

This study rejuvenates human amniotic fluid stem cells to generate induced mesenchymal stem cells with enhanced therapeutic potential through epigenetic reprogramming.

DOI 10.1101/2025.03.25.645040

humanexperimental assay

𝕏 PostBluesky

Cell BiologyBiology OpenPeer-reviewedOct 29, 2024

Disrupting the interaction between AMBRA1 and DLC1 is a promising therapeutic strategy for neurodegeneration that prevents apoptosis while enhancing autophagy and mitophagy

Kate Hawkins, Meg Watt, Sébastien Gillotin…·Janet Brownlees

The paper investigates AMBRA1 mutants to identify strategies that prevent apoptosis and enhance autophagy/mitophagy in neurodegeneration, finding that AMBRA1TAT, which disrupts DLC1 interaction, shows the most promise.

4 citations

humanmouse

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