Author

William J. Kissner

University of North Carolina at Chapel HillORCID 0000-0001-7335-7877Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

David B. HilliD· 2 shared Matthew R. MarkovetziD· 2 shared Alexandra F. FreemaniD· 1 shared Anshulika Deshmukh· 1 shared Barbara R. GrubbiD· 1 shared Brian M. ButtoniD· 1 shared Camille EhréiD· 1 shared Charles R. EstheriD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCell Biology

David B. HillORCID

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2 papers

Recent·Most cited

Cell BiologyAmerican Journal of Respiratory and Critical Care MedicinePeer-reviewedJun 13, 2025

Dysregulated Airway Host Defense in Hyper IgE Syndrome due to STAT3 Mutations

Ling Sun, Samantha A. Walls, Hong Dang…·Gang Chen

This study identifies airway epithelial host defense defects caused by STAT3 mutations in Hyper IgE Syndrome, contributing to chronic pulmonary infection.

7 citations

humanbulk RNA-seq

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MicrobiologyPLoS ONEPeer-reviewedFeb 23, 2024

Combination Treatment to Improve Mucociliary Transport of Pseudomonas aeruginosa Biofilms

Kaitlyn R. Rouillard, Christopher P. Esther, William J. Kissner…·David B. Hill

This paper tests combination treatments to disrupt Pseudomonas aeruginosa biofilms in mucus, finding that NP40, hyaluronic acid, and tobramycin most effectively increase mucociliary transport and reduce bacterial viability.

8 citations

humanexperimental assay

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